To discover the core gene expression features of ovarian cancer spheroids
No associated publication
Specimen part, Cell line
View SamplesTo discover the core gene expression features of CtBP1, CtBP2 differently regulated in ovarian cancer SKOV3 cells.
No associated publication
Specimen part, Cell line, Treatment
View SamplesTo discover the abnormal regulatory role of Pin1 in the ovarian cancer, The Pin1 full cDNA was knockIn (KI) the normal ovarian epithelium (Hose), and parallelly Pin1 was knockdown in the Ovarian cancer cells MCAS and sk-ov-3, and then compared the expression profiles of them to discovery the key features regulated by Pin1.
No associated publication
Cell line
View SamplesThe objective of this study is to determine how inhibition of microRNA 10b affects gene expression in neurospheres cultures of glioblastoma stem-like cells.
Therapeutic potential of targeting microRNA-10b in established intracranial glioblastoma: first steps toward the clinic.
Treatment
View SamplesWe used microarray analysis to identify differences in gene expression levels, in liver and in quadriceps skeletal muscle, between 18h (overnight) fasted WT control and Kruppel-like factor 15 (KLF15)-null mice.
Regulation of gluconeogenesis by Krüppel-like factor 15.
No sample metadata fields
View SamplesOncogenic tyrosine kinases, such as BCR-ABL, TEL-ABL, TEL-PDGF-beta-R and FLT3-ITD, play a major role in the development of hematopoietic malignancy. They activate many of the same signal transduction pathways.
Id1 is a common downstream target of oncogenic tyrosine kinases in leukemic cells.
No sample metadata fields
View SamplesComparative analysis of gene expression patterns between prenatal (embryonic e16d) and postnatal (p1d and p7d) mouse kidney
No associated publication
Age, Specimen part
View SamplesTGF-beta3 produced by developing Th17 cells induces highly pathogenic T cells that are functionally and molecularly distinct from TGF-beta1-induced Th17 cells. The microarray data represent a distinct molecular signature for pathogenic versus non-pathogenic Th17 cells.
Induction and molecular signature of pathogenic TH17 cells.
Sex, Specimen part
View SamplesThis SuperSeries is composed of the SubSeries listed below.
PRC2 loss amplifies Ras-driven transcription and confers sensitivity to BRD4-based therapies.
Cell line, Treatment
View SamplesmTOR and HDAC inhibitors induce cell death of malignant peripheral nerve sheath tumors (MPNSTs) in vitro, and in vivo
mTOR and HDAC Inhibitors Converge on the TXNIP/Thioredoxin Pathway to Cause Catastrophic Oxidative Stress and Regression of RAS-Driven Tumors.
Cell line, Treatment
View Samples