This SuperSeries is composed of the SubSeries listed below.
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Specimen part, Cell line, Treatment
View SamplesComparison of laminin binding and laminin non-binding germ cells
Defining the spermatogonial stem cell.
No sample metadata fields
View SamplesRat germ cells
Defining the spermatogonial stem cell.
No sample metadata fields
View SamplesAnalysis of the influence of celiac disease-associated bacteria and gluten on intestinal epithelial cells
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Cell line, Treatment
View SamplesAnalysis of the influence of celiac disease-associated bacteria and gluten on intestinal epithelial cells
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Cell line, Treatment
View SamplesAnalysis of the influence of celiac disease-associated bacteria on intestinal epithelial cells
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Cell line, Treatment
View SamplesAnalysis of role of small intestinal intraepithelial lymphocytes (IELs) in the immunopathology of celiac disease
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Specimen part
View SamplesAnalysis of role of small intestinal epithelial cells (IECs) in the immunopathology of celiac disease
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Specimen part
View SamplesPseudomonas aeruginosa is a virulent opportunistic pathogen responsible for high morbity in COPD, burns , implanted medical devices and cystic fibrosis.
Anr and its activation by PlcH activity in Pseudomonas aeruginosa host colonization and virulence.
No sample metadata fields
View SamplesExposure to high levels of arsenic in drinking water is associated with several types of cancers including lung, bladder and skin, as well as vascular disease and diabetes. Drinking water standards are based primarily on epidemiology and extrapolation from higher dose experiments, rather than measurements of phenotypic changes associated with chronic exposure to levels of arsenic similar to the current standard of 10ppb, and little is known about the difference between arsenic in food as opposed to arsenic in water. Measurement of phenotypic changes at low doses may be confounded by the effect of laboratory diet, in part because of trace amounts of arsenic in standard laboratory chows, but also because of broad metabolic changes in response to the chow itself. Finally, this series contrasts 8hr, 1mg/kg injected arsenic with the various chronic exposures, and also contrasts the acute effects of arsenic, dexamethasone or their combination. Male C57BL/6 mice were fed on two commercially available laboratory diets (LRD-5001 and AIN-76A) were chronically exposed, through drinking water or food, to environmentally relevant concentrations of sodium arsenite, or acutely exposed to dexamethasone.
Laboratory diet profoundly alters gene expression and confounds genomic analysis in mouse liver and lung.
No sample metadata fields
View Samples