Analysis of the genes and cellular signalling cascades mediating the response of SCN slices to vasoactive intestinal peptide (VIP). Primary goal was to find novel genes that may be involved in circadian phase shifting for further study. Promoter analysis of significantly regulated genes and gene ontology analysis would provide information into pathways VIP acts through in the SCN.
Vasoactive intestinal peptide controls the suprachiasmatic circadian clock network via ERK1/2 and DUSP4 signalling.
Specimen part
View SamplesThis SuperSeries is composed of the SubSeries listed below.
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Specimen part, Cell line, Treatment
View SamplesComparison of laminin binding and laminin non-binding germ cells
Defining the spermatogonial stem cell.
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View SamplesRat germ cells
Defining the spermatogonial stem cell.
No sample metadata fields
View SamplesAnalysis of the influence of celiac disease-associated bacteria and gluten on intestinal epithelial cells
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Cell line, Treatment
View SamplesAnalysis of the influence of celiac disease-associated bacteria and gluten on intestinal epithelial cells
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Cell line, Treatment
View SamplesAnalysis of the influence of celiac disease-associated bacteria on intestinal epithelial cells
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Cell line, Treatment
View SamplesAnalysis of role of small intestinal intraepithelial lymphocytes (IELs) in the immunopathology of celiac disease
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Specimen part
View SamplesAnalysis of role of small intestinal epithelial cells (IECs) in the immunopathology of celiac disease
Immunopathology of childhood celiac disease-Key role of intestinal epithelial cells.
Specimen part
View SamplesPseudomonas aeruginosa is a virulent opportunistic pathogen responsible for high morbity in COPD, burns , implanted medical devices and cystic fibrosis.
Anr and its activation by PlcH activity in Pseudomonas aeruginosa host colonization and virulence.
No sample metadata fields
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