Description
Alcoholic liver disease is a pathological condition caused by over-consumption of alcohol. Due to the high prevalence of morbidity and mortality associated with this disease, there remains a need to elucidate the molecular mechanisms underlying the etiology to develop new treatments. Since peroxisome proliferator-activated receptor-/ (PPAR/) modulates ethanol-induced hepatic effects, the present study examined alterations in gene expression that may contribute to this disease.